Summary:
Possible mechanisms by which lung bacteria may interact with host factors and contribute to the pathogenesis and progression of idiopathic pulmonary fibrosis (IPF). MUC5B: mucin 5B; TOLLIP: Toll-interacting protein; AEC: alveolar epithelial cell. Reproduced and modified from [34] with permission.
Type:
Figure
Sub Component:
Normal
Slug:
F22
Highwire: Type:
fragment
Highwire: Parent:
HighWire: Journal/Corpus Code:
ersbk
Highwire: pisa_id:
ersbk;9781849841023/1/chapter_10/F22
Highwire: pisa_master:
ersbk;9781849841023/1/chapter_10/F22
HighWire: Atom Path:
/ersbk/9781849841023/9781849841023/SEC14/F22.atom
Highwire: cpath:
/content/9781849841023/9781849841023/SEC14/F22
Image - Large:
Highwire: cpathalias:
/content/ersbk/9781849841023/9781849841023/SEC14/F22
Image - Medium:
Highwire: Variants:
expansion
Image - Small:
Highwire: State:
Released
Contributors:
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